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An advanced form of fatty liver involving inflammation and liver damage—often driven by excess fructose metabolism.
Fructose metabolism plays a central role in NASH by rapidly generating fat, uric acid, and oxidative stress in liver cells. Over time, this leads to inflammation, scarring, and liver damage—even in the absence of alcohol. And because the polyol pathway can produce fructose endogenously, this damage can occur even on a sugar-free diet.
SugarShield is formulated to help support liver resilience by modulating the effects of excess fructose metabolism. It’s a science-driven approach to protecting cellular energy and metabolic health—before inflammation takes hold.
Non-Alcoholic Steatohepatitis (NASH) is a severe progression of non-alcoholic fatty liver disease (NAFLD), characterized not just by fat buildup but also by liver inflammation and cellular damage.
NASH may be silent in early stages but can lead to:
- Liver fibrosis and scarring
- Cirrhosis
- Liver failure or cancer
It's a rising cause of liver transplants worldwide.
Fructose metabolism plays a key role in NASH:
- Promotes fat accumulation via de novo lipogenesis
- Elevates uric acid and oxidative stress
- Disrupts mitochondrial energy, triggering inflammation
Even without sugar, endogenous fructose from carbs, alcohol, or salt can continue to fuel the NASH process—especially in metabolically compromised individuals.
At LIV3, we see NASH as a downstream effect of a broken energy pathway—and targeting fructokinase is a proactive step to prevent or reverse this damage at its metabolic root."
Scientists have successfully restored the lost uricase enzyme, a key breakthrough in combating fructose-induced fat formation. This discovery offers new hope for preventing obesity and metabolic disorders by targeting how the body processes sugar and stores fat.
Fructose intolerance isn’t just a gut issue—it’s an energy problem. This article explains how limited fructose absorption in the intestine and excess cellular metabolism of fructose are two sides of the same process, and how restoring fructose metabolism can repair both gut and metabolic health.
Perimenopause can disrupt metabolism, and fructose may make it worse. This blog uncovers how fructose affects hormonal balance, weight gain, and energy—and how to support your body naturally.
Luteolin is a rare plant flavonoid shown to naturally quiet the fructose pathway—the same metabolic switch targeted by pharmaceutical fructokinase inhibitors. By protecting cellular energy and lowering uric acid and inflammation, it helps restore the stable, high-efficiency metabolism that modern diets have lost.
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